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細胞性免疫と生体防御
https://doi.org/10.34429/00000853
https://doi.org/10.34429/00000853a997964e-d508-47b5-b18f-51d1e8551b74
| 名前 / ファイル | ライセンス | アクション |
|---|---|---|
KJ00000561012.pdf (659.6 kB)
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| Item type | 紀要論文 / Departmental Bulletin Paper(1) | |||||||
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| 公開日 | 2014-08-14 | |||||||
| タイトル | ||||||||
| タイトル | 細胞性免疫と生体防御 | |||||||
| 言語 | ja | |||||||
| 言語 | ||||||||
| 言語 | jpn | |||||||
| キーワード | ||||||||
| 言語 | ja | |||||||
| 主題Scheme | Other | |||||||
| 主題 | 感作リンパ球 | |||||||
| キーワード | ||||||||
| 言語 | ja | |||||||
| 主題Scheme | Other | |||||||
| 主題 | 結核菌 | |||||||
| キーワード | ||||||||
| 言語 | ja | |||||||
| 主題Scheme | Other | |||||||
| 主題 | 細胞性免疫 | |||||||
| キーワード | ||||||||
| 言語 | ja | |||||||
| 主題Scheme | Other | |||||||
| 主題 | 細胞障害性T細胞 | |||||||
| キーワード | ||||||||
| 言語 | ja | |||||||
| 主題Scheme | Other | |||||||
| 主題 | ウイルス感染細胞 | |||||||
| 資源タイプ | ||||||||
| 資源タイプ識別子 | http://purl.org/coar/resource_type/c_6501 | |||||||
| 資源タイプ | departmental bulletin paper | |||||||
| ID登録 | ||||||||
| ID登録 | 10.34429/00000853 | |||||||
| ID登録タイプ | JaLC | |||||||
| タイトルヨミ | ||||||||
| ja-Kana | ||||||||
| サイボウセイ メンエキ ト セイタイ ボウギョ | ||||||||
| タイトル(別表記) | ||||||||
| その他のタイトル | Cell-mediated immunity in host defense mechanisms | |||||||
| 言語 | en | |||||||
| 著者名 |
深沢, 義村
× 深沢, 義村
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| 著者名(別表記) | ||||||||
| 識別子Scheme | WEKO | |||||||
| 識別子 | 5001 | |||||||
| 姓名 | Fukazawa, Yoshimura(en) | |||||||
| 内容 | ||||||||
| 内容記述タイプ | Other | |||||||
| 内容記述 | Cell-mediated immunity plays a major role in the resistance of host to many bacteria, fungi, and protozoa, all of which have the capacity to live and multiply in the mononuclear phagocytes. Cell-mediated immune mechanisms can be functioned by collaboration with helper T cells (Th), cytotoxic T cells (Tc, CTL), macrophages and natural killer cells. Tdth that funcions as effector cells for delayed-type hypersensitivity reaction, is generated from Th, of which surface antigen is termed as L3T4 for mouse and CD 4 for human, in response to antigenic stimulus. One of lymphokines, macrophage activating factor (MAF) which is now referred to γ-interferon (IFN-γ) , is secreted from Tdth, and activate macrophage for increased capacity for microbicidal activity and tumor cytotoxicity. CTL with specificity for viral antigen is generated that inhibit the virus from propagating by killing the infected host ells as well as by secreting IFN-γ. CTL is generated also during tumor growth, and allograft rejection. In graft rejection, CTL is directed mainly to class I alloantigens which serve as the main stimulus for its generation. In contrast, Th is reacting mostly with the class II alloantigens found in cells of the graft. CTL plays roles for destruction of tumor cells and allograft by adhering to target cells. CTL function is restricted in major histocompatibility complex (MHC) products and is collaborated with Th and macrophages. | |||||||
| 言語 | en | |||||||
| 出版者 | ||||||||
| 出版者 | 山梨医科大学 | |||||||
| 言語 | ja | |||||||
| ISSN | ||||||||
| 収録物識別子タイプ | ISSN | |||||||
| 収録物識別子 | 0910-5069 | |||||||
| NCID | ||||||||
| 収録物識別子タイプ | NCID | |||||||
| 収録物識別子 | AN10032951 | |||||||
| 書誌情報 |
ja : 山梨医科大学紀要 en : Bulletin of Yamanashi Medical University 巻 4, p. 8-15, 発行日 1987 |
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| 著者版フラグ | ||||||||
| 出版タイプ | VoR | |||||||
| 出版タイプResource | http://purl.org/coar/version/c_970fb48d4fbd8a85 | |||||||
